Impaired mechanisms of homeostatic plasticity may drive cortical hyperexcitability in a mouse model of Fragile X Syndrome All Restricted

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About this Dissertation

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School	Laney Graduate School
Department	Biological and Biomedical Sciences
Subfield / Discipline	Neuroscience
Degree	Ph.D.
Submission	Dissertation
Language	English
Research Field	Biology, Neuroscience Biology, Cell Biology, Physiology
Parola chiave	processing bodies Fragile X Syndrome Intrinsic excitability mitochondria homeostatic plasticity Fragile X Mental Retardation Protein
Committee Chair / Thesis Advisor	Victor Faundez, Emory University Gary J. Bassell, Emory University
Committee Members	Robert Liu, Emory University Peng Jin, Emory University Matt Rowan, Emory University

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