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processing bodies
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Impaired mechanisms of homeostatic plasticity may drive cortical hyperexcitability in a mouse model of Fragile X Syndrome
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Year
Spring 2021
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School
Laney Graduate School
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Department
Biological and Biomedical Sciences
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Degree
Ph.D.
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Submission Type
Dissertation
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Research Field
Biology, Cell
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Biology, Neuroscience
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Biology, Physiology
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Committee
Gary J. Bassell, Emory University
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Matt Rowan, Emory University
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Peng Jin, Emory University
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Robert Liu, Emory University
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Victor Faundez, Emory University
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Keyword
Fragile X Mental Retardation Protein
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Fragile X Syndrome
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Intrinsic excitability
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homeostatic plasticity
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mitochondria
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