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processing bodies
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1.
Impaired mechanisms of homeostatic plasticity may drive cortical hyperexcitability in a mouse model of Fragile X Syndrome
Student Name:
Buelow, Pernille
Date Uploaded:
03/03/2021
Research Field:
Biology, Neuroscience, Biology, Cell, and Biology, Physiology
Department:
Biological and Biomedical Sciences
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Year
Spring 2021
1
School
Laney Graduate School
1
Department
Biological and Biomedical Sciences
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Ph.D.
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Submission Type
Dissertation
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Research Field
Biology, Cell
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Biology, Neuroscience
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Biology, Physiology
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Gary J. Bassell, Emory University
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Matt Rowan, Emory University
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Peng Jin, Emory University
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Robert Liu, Emory University
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Victor Faundez, Emory University
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Keyword
Fragile X Mental Retardation Protein
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Fragile X Syndrome
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Intrinsic excitability
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homeostatic plasticity
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mitochondria
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