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Molecular and Genetic Analysis of Mitochondrial Phenotypes in
22q11.2 Microdeletion Syndrome, a Neurodevelopmental Disorder.
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Analysis of Dysbindin-BLOC-1 Complex Mechanisms in the
Neuromuscular Synapse of the Fruit Fly Drosophila melanogaster
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Studies of Vesicle Trafficking Following Functional Depletion of
Clathrin: Use of Clathrin Light Chain-FKBP-Binding Domain
Chimera
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Hermansky-Pudlak Complexes, AP-3 and BLOC-1, Regulate the MolecularArchitecture of the Synapse
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Targeting of the Lysosomal Tether, The HOPS Complex,
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Regulation of Drosophila Synaptic Function and Plasticity by a
Schizophrenia Susceptibility Network.
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Actin Cytoskeleton Regulators Interact with the
Hermansky-Pudlak Syndrome Complex BLOC-1 and its Cargo
Phosphatidylinositol-4-kinase Type II Alpha
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Golgi-Dependent Mechanisms of Cellular Copper Homeostasis
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Interactions Between Mitochondrial Transporters and the Protein Synthesis Machinery in 22q11.2 Deletion Syndrome
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